By Frank J. Dixon

ISBN-10: 0120224550

ISBN-13: 9780120224555

From the experiences of past Volumes "Provides unrivalled worth in either educational and financial phrases and may be bought by means of not easy pressed librarians as a huge precedence to be jealously defended." -JOURNAL OF scientific MICROBIOLOGY "Advances in Immunology needs to locate itself one of the so much energetic volumes within the libraries of our universities and institutions." -SCIENCE "A provocative and scholarly evaluate of research." -JOURNAL OF the yank scientific organization "Deserves an everlasting position in biomedical libraries as an relief in examine and in teaching." -JOURNAL OF IMMUNOLOGIC equipment

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1992a) have proposed that exposure to SCF may first result in monovalent binding of the ligand to the SCFR, followed by the production of an intermediate receptor dimer bound by one arm of the ligand molecule. , 1992). This work has determined that human SCF binds primarily to the first two Ig-like domains of the amino-terminal half of the SCFR ectodomain, but that structural determinants distal to this region may also contribute to ligand recognition. One of the elements involved in both SCF-dependent and CSF-1dependent signal transduction may be encoded at or regulated by the mi locus on mouse chromosome 6 (Silvers, 1979b).

Because the receptors for PDGF and CSF-1 exhibit structural homology with the c-kit receptor, Tsai et al. (1993) investigated whether early response genes were induced by the stimulation of the c-kit receptors of the mouse bone marrow derived-cultured mast cells. Tsai 36 STEPHEN J. GALL1 ET AL. et al. (1993) found that SCF-dependent stimulation of the SCF receptors in BMCMCs can lead to increased levels of mRNAs for the c-fos gene and for the genes of the j u n family. These findings thus demonstrated that the induction of these protooncogenes represents one of the immediate responses of at least one cell type, the mast cell, to activation of the SCFR by its ligand.

1991a), the results may have reflected effects of SCF/SCFR function on mast cell survival and/or proliferation, rather than on maturation per se. , 1991b). It had been shown by Levi-Schaffer et al. (1986) that coculture of immature mouse mast cells on 3T3 fibroblasts could induce the mast cells to acquire a more mature phenotype, including increased histamine content, induction of the ability to synthesize and store substantial amounts of heparin, and alteration of the histochemical characteristics of the cells.

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Advances in Immunology, Vol. 55 by Frank J. Dixon

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